Article: Data from I. Touitou et al provide new insights into clinical rheumatology.

According to recent research from Montpellier, France, "Auto inflammatory diseases (AIDs) are illnesses caused by primary dysfunction of the innate immune system. Proteins that are mutated in AIDs mediate the regulation of NF kappa B activation, cell apoptosis, and IL-1 beta secretion through cross-regulated and sometimes common signaling pathways."

"AIDs include a broad number of monogenic (e.g., familial Mediterranean fever (FMF), cryopyrin-associated periodic syndrome (CAPS), mevalonate kinase deficiency (MKD), tumor necrosis factor (TNF)-receptor-associated periodic syndrome (TRAPS)] and multifactorial (e.g., Behcet's syndrome) disorders. These conditions are ...

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