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Recurrence of primary hyperoxaluria: An avoidable catastrophe following kidney transplant
- Article from:
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Journal of Postgraduate Medicine
- Article date:
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July 1, 2008
- Author:
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Copyright informationCopyright Medknow Publications Jul-Sep 2008. Provided by ProQuest LLC. (Hide copyright information)
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Primary hyperoxaluria (PHO) Types 1 and 2 are rare autosomal recessive disorders, characterized by oxalate overload due to deficiency of the liver enzymes alanine glyoxalate aminotransferase (AGT) and D-glycerate dehydrogenase respectively. [1] Type 1 PHO occurs in 0.11 to 0.26 per 100,000 births and results in severe disease, while there are less than 30 reported cases of the less severe Type 2 PHO. [2] The excess body oxalate in Type 1 PHO is excreted in urine resulting in nephrocalcinosis, nephrolithiasis and is also deposited in multiple organs. Primary hyperoxaluria is a "nephrologic liver disease" hence, combined liver and kidney transplant is the treatment of choice. [2],[3] Isolated ...
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